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This modified peptide that lacked Tyr at residue 10 would be predicted not to lead to cell death or protein oxidation of neuronal cultures or be able to activate the nitrone spin trap to form the EPR-measurable nitroxide free radical if the Tyr residue of Aβ42 was critical to these effects of Aβ42
كريم ببولي جلوتاميك اسيد
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