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Acne vulgaris (commonly called acne) provides a promising disease model to study the interactions between the host and the skin microbiota in disease pathogenesis, because the microbiota is less complex and the disease has been associated with a single dominant bacterium, Propionibacterium acnes ( 2, 6–8 )
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12-h PKCδ/CD38 inhibitors (CD38 inhibitor 1, 20 nM) pre-treatment, HOKs were stimulated with LPS or activated CD4+ T cells production for 24 h
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