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Energy metabolism and mitochondrial dynamics in neurodegenerative disorders: focus on hyperhomocysteinemia and neonatal hypoxia-ischemia - ScienceDirect
Ferroptosis is morphologically, biochemically, and genetically distinct from necrosis, apoptosis, and autophagy, and these differing features include abnormal mitochondrial membrane density, iron accumulation, lipid peroxidation, overexpression of ferroptosis biomarkers, and death of leucocyte subsets and the corresponding loss of immune function [11, 18,19,20,21]
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