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By avoiding established pharmacological treatments that may produce significant side effects, parents try to alleviate behavioral problems and associated comorbid conditions using alternative treatments
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Evidently, intracellular cyst(e)ine contribute to ferroptosis suppression via at least two additional pathways in different contexts: in a mouse lung adenocarcinoma model, NFS1 cysteine desulfurase utilizes cysteine to synthesize iron–sulfur clusters, which are necessary for preventing activation of the iron-starvation response, subsequent iron influx and ferroptosis 14 (Fig
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