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To remove senescent cells, these treatments induce apoptosis in SCs by inhibiting anti-apoptotic pathways mediated by Bcl-2 family proteins (e.g., Bcl-2 and Bcl-xL) and reduce the senescence-associated secretory phenotype (SASP) within senescent chondrocytes and synovial cells within the joint microenvironment, as shown in Fig

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[DOI] [PubMed] [Google Scholar] 126.Coffman FD and Dunn MF, Insulin—Metal Ion Interactions: The Binding of Divalent Cations to Insulin Hexamers and Tetrarners and the Assembly of Insulin Hexamers, Biochemistry, 1988, 27, 6179–6187

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