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Another reason was that, although a concentration gradient between extracellular and intracellular Acr formed, the passive diffusion of Acr across the cell membrane might heavily rely on the transport capacity of aquaglyceroporin FPS1, the loss of which confers resistance to Acr in S

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Further, GSTP1 attenuated autophosphorylation of apoptosis signal-regulating kinase 1 (ASK1) and inhibited TRAF2-ASK1-induced apoptosis [58]