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A decrease in pro-inflammatory cytokines was observed in preliminary human studies (41, 45), with a decrease in both proinflammatory cytokines and microglia activation noted in murine models (4, 42, 45–47)
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Tumors exhibit suppressed gluconeogenesis but up-regulate distinct Nfrf2 target genes A recent study (Gu et al, 2025) showed that during the progression from metabolic dysfunction–associated steatohepatitis (MASH) to HCC, FBP1 expression declines in premalignant hepatocytes
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