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Thus agents that exploit mutations or amplification of the cKIT gene found in gastrointestinal stromal tumors, or the EGFR found in non-small cell lung cancers, or the human epidermal growth receptor2 (HER2/NEU) oncogene in breast cancer, or the BCR-ABL translocation protein product in chronic myelogenous leukemia (CML) have become important components in the therapy of these diseases (see Chap