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found that MEL can regulate neuroinflammation through activation of the Nrf2 pathway (136), another experimental and clinical study confirmed the protective effect of MEL on glial cells and its beneficial effect on DPN, but the specific therapeutic regimen of administration needs to be further clarified (137)

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It was observed that in a subgroup of FM patients, treatment with N-methyl-D-aspartate (NMDA) receptor antagonists improved symptoms and pain perception [22]