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glutathione receptor t cell system enhancement for cardiac protection: pharmacological options against oxidative stress and ferroptosis Hi-Tech
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glutathione receptor t cell system enhancement for cardiac protection: pharmacological options against oxidative stress and ferroptosis Hi-Tech
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glutathione receptor t cell system enhancement for cardiac protection: pharmacological options against oxidative stress and ferroptosis Hi-Tech
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When ROS production increases (due to ageing, microbial infections, extensive exercise, or pollutants/toxins [93] such ionizing and UV radiation, pesticides, and ozone, etc.) and antioxidant defences are overwhelmed, ROS can damage mitochondrial lipids (especially cardiolipin in the inner membrane) and mitochondrial DNA and inactivate ETC proteins
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doi: 10.1016/j.ebiom.2018.09.015
glutathione receptor t cell system enhancement for cardiac protection: pharmacological options against oxidative stress and ferroptosis Hi-Tech
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