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Compared to other ROS, O 2 ·− is considered a moderately reactive radical with a short half-life and low mobility due to its negative charge, which limits its ability to cross biological membranes (Demidchik 2015)

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This pathological ROS overproduction is a common final pathway in various forms of acquired hearing loss, including noise-induced and drug-induced (cisplatin) hearing impairment, and current evidence from human studies supporting antioxidant-based interventions remains insufficient (Teraoka et al., 2024)