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glutathione reductase with nadph steady state NOX-like ROS production by reductase: iScience BPC DELAYED
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glutathione reductase with nadph steady state NOX-like ROS production by reductase: iScience BPC DELAYED
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glutathione reductase with nadph steady state NOX-like ROS production by reductase: iScience BPC DELAYED
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glutathione reductase with nadph steady state NOX-like ROS production by reductase: iScience BPC DELAYED
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4.2.3 Bromodomain and extraterminal domain (BET) inhibitors In the treatment of malignant tumors, bromodomain (BRDs) and extraterminal (BET) proteins inhibitors have been appeared to be novel targeted medicines that control the epigenetic alterations in such conditions (Filippakopoulos et al., 2010)
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9–11,13,14 Oxidative stress compounded with lysosomal iron uptake within the mitochondria leads to mitochondrial membrane dysfunction via disruption of the mitochondrial membrane permeability transition pore, triggering cell necrosis
glutathione reductase with nadph steady state NOX-like ROS production by reductase: iScience BPC DELAYED
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