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LPLs are commonly formed when the ester bonds of PLs are enzymatically hydrolysed by phospholipase A, producing LPL and a free fatty acid chain [41]
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The possibility that BPC 157 therapy might affect many mechanisms of secondary neuronal injury [1] was supported by the immediate events occurring upon BPC 157 application at 10 min post-injury, as well as at day 4 after injury
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