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This gives rise to the intriguing possibility that, if intracellular levels of GSH are sufficient to mitigate the inhibitory action of the metabolites, Me-DDTC-SO may perhaps be a better candidate for specific inhibition of ALDH2, given its ability to overcome GSH scavenging (Mays et al., 1998)

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In both groups, with similar cumulative BTZ doses, the regimen administered at a lower intensity resulted in a shorter recovery time (2.3 vs